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- BBB DamageCell Death and ApoptosisNeuroinflammationNeuronal Damage and RepairOxidative StressVascular Mechanisms
- 3D BBB Models
- Organ-on-a-Chip BBB ModelsTranswell Models3D BBB Models Culture Substrate
- 3D HydrogelsCollagensAnmial BBB ModelsCell Lines for BBB Modeling
- Immortalized CellsPrimary CellsStem Cells
Neuroinflammation
Neuroinflammation is a key component of stroke pathology and involves the activation of immune cells such as microglia and astrocytes, and the release of inflammatory molecules such as cytokines and chemokines. Antibodies that target components of the neuroinflammatory pathway can provide valuable insights into the underlying mechanisms of stroke and may also serve as potential therapeutic targets, thus developing targeted therapies that can modulate this response and improve outcomes for stroke patients.
Please access our full range of neuroinflammation-related antibodies for stroke research or contact our scientist for personalized assistance.
Search Under Neuroinflammation
References- Qin C, Yang S, Chu YH, et al. Signaling pathways involved in ischemic stroke: molecular mechanisms and therapeutic interventions. Signal Transduct Target Ther. 2022, 7(1):215.
- Sierra C, Coca A, Schiffrin EL. Vascular mechanisms in the pathogenesis of stroke. Curr Hypertens Rep. 2011,13(3):200-7.
All of our products are intended for preclinical research use only and cannot be used to diagnose, treat or manage patients.
Ace Therapeutics is a global leading provider of stroke research services. We are committed to accelerating progress in stroke research and drug development.
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